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Domestic Violence and Abuse

How UV Rays Damage Skin

People diagnosed with skin cancer often ask
– why did I get skin cancer, and what can I do to prevent my children from getting skin
cancer? Those are excellent questions. For you to know how to prevent skin ageing
and skin cancer, you need to know about the dark side of the sun. Today I want to share with you a deep dive
into how the sun can accelerate skin ageing and also increase the risk of skin cancer. When sunlight reaches the earth’s surface,
it is made up of three main types of radiation. Infrared radiation is what gives sunlight
its warmth. Visible light helps us see. And then there’s UV light – a types of
radiation that we can’t feel or see. When it comes to the damaging effects of sunlight,
UV is a major culprit. There are 2 types of UV that reach us on earth’s
surface: UVA and UVB. Both types of UV can damage the skin, but
in slightly different ways. Of the two, UVB is the most damaging form
of UV radiation – it causes sunburns and increases the risk of skin cancer. The upper layer of the skin is called the
epidermis. Visible light from the sun is reflected off
the epidermis, but UVB penetrates into it. In the epidermis, UVB reacts with the DNA
of cells and causes DNA damage. Our skin cells are able to repair DNA damage,
but with time, if enough DNA damage accumulates, skin cancer can form. In the epidermis, there are cells called melanocytes. When skin cells are damaged by UV, melanocytes
release a dark pigment called melanin. Melanin is able to absorb UV light, and so
the body uses melanin as a kind of UV shield. But melanin isn’t able to completely stop
skin damage from UVB. A skin tan is actually a sign that skin damage
has already occurred, and the body is preparing itself for further UV exposure. In a sunburn, UVB causes so much damage that
the skin cells die and the upper layer of the skin peels off. The death of cells also triggers inflammation,
making the skin red, hot and painful to touch. UVA can also increase the risk of skin cancer
but mostly it is involved in accelerating skin ageing. UVA is able to infiltrate down to the deep
layer of the skin, called the dermis. The dermis has a scaffolding of molecules
such as collagen that makes your skin elastic. When you pull on your skin, it is this scaffolding
that helps your skin spring back. UVA damages this collagen scaffolding. This makes your skin more prone to wrinkling
and sagging. It’s not all bad news though. We know that sunlight in moderate amounts
is helpful for making Vitamin D and that’s what I talked about in my last video. Also, UV light prescribed by doctors can be
helpful for immune based skin conditions like psoriasis. So in healthy doses, sunlight can be good
for you, but how do you protect yourself from the damaging effects of excess UV. Well in my next video, I’m going to share
with you my 4 step process to help you choose the right sunscreen for your skin. Thanks for watching and I’ll see you in
the next one

Cesar Sullivan

7 thoughts on “How UV Rays Damage Skin

  1. Hello. I have a question about the still unexplored "impact of UV and UVC radiation on the skin or the human body. I have a curious experience that, in contrast to my gene, could have caused a great deal of impact. Made in the 80s. The exposure occurred in 2006. It is possible that hot flushes in the face, still red hypersensitive eyes and redness is a consequence? I had a lamp on the table about 1m from the face for about 25 minutes. my eyesight was narrowed, I wasn't feeling warm, I was red on the next morning and my eyes hurt in the morning after the exposure, they gave me panthenol in the emergency room. I see an increase in blood supply, including eyes, I am still red, I am very red at any activity. accusations, hotness and hot flashes make you annoyed by eating, and your eyes turn red and the whole face gets blood flowing into extreme colors. Is it possible that this is due to UVC burns or is it just a coincidence and is it a hormonal or other matter? So I am a light type, so if there was DNA damage at the cellular level (as reported in available UVC resources), could it be the change? How strong must be the radiation and the exposure time to make this possible? Thank you very much for your answer.

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